Insulin Resistance

When the glycogen level in the muscle cells is full, the cells become insulin resistant, that is a higher insulin level is required to push glucose in to the cell. OK. So, too much glucose results in high insulin. The cell is repleted of glucose, plain full.

So exercise, vigorous or strenuous burns up glucose and glycogen, and the cell will accept glucose again at lower insulin level. The cell is partly depleted of glucose. Gentle exercise tents to burn more fat.


Now remember the fat cell, with a input hormone and an output hormone, both keying off the absolute insulin level. The input is turned on by high insulin, putting fat into fat cells, as well as glucose. The output requires the insulin level to be low to release FFA (free fatty acid) for energy. So if we are insulin resistant, we never get to have low insulin enough to release FFA. We conserve fat, not a good thing for weight loss.

The insulin also keeps the blood triglycerides in the chylomicrons, and we get that craving for food.  There is a relationship between insulin and chylomicrons. Insulin resistance results in to much insulin, and FFA is trapped in chylomicrons and fat cells.

So, with glucose full and no FFA, we get hunger unless we do something to cause the glucose to be burned and release FFA and A co A for the mitochondria, so they are calling feed me, feed me, and we get a severe food craving, not hunger.

So snacks need to be pure fat, and a vector,not carbohydrates. That allows the craving to be resolved without raising the insulin level.