Oiling the gut

Oiling the gut as per Shangri La seem to delay the onset of hunger. Let me make this clear, it does not increase satiation noticeably, but allows me to go longer between eating without hunger. Once hunger is there, look out, it is too late.

This is a N=1 experiment only that I have been try in the past 5 days. I have been mixing canola oil and a bit of honey as an emulsifying agent with water and drinking it down, as gross as it is. About a table spoon of oil, 15 grams, and a glob of honey with 3/4 liter of water, ice chips, and shake well. Old one kg peanut butter jar, just because it was there. I have been having a big slug, ever 3-2 hours, with small oil rich meals.

I think that Seth Roberts has an incomplete explanation as to why it works. First, is it often said that hunger is a gut physical sensation, and that bit of oil may provide a first pass effect, feeding the gut. It does seem to change that active hunger, and ease the amount of food required for satiation a bit, but only if I eat before I get hungry. I am also trying a shot before meals, when practical.

The energy level is much higher on oil than before. My normal diet is a low carb calorie restricted diet, even to maintain weight, as I will overeat on anything. I expect that nature did not want us to be high energy and using body fat only. That would not be prudent. Slow release of saturated fat may be the energy governor. I can be still high energy when required, but the motivation required must be much more extreme. Yet walking at a slow/reasonable pace does not require pushing myself.

This oiling may also be speeding ketone production, or just provide more oil to be converted to ketones. The mono and pufa oils are not used for fat production, according to LR. Vanderhaeghe et al., but she thing saturated fat causes obesity, so it is not a trusted source. It is saturated fat, but it is the saturated fat that we make that is the big source. I do think that she may be right, where we were eating low carb and getting fat, however rare that might be.

What Causes excess hunger?

Remaining hungry, needing to be hungry to loss weight, or even to maintain weight loss is not really practical long term, but that is what I am faced with.

We have heard at length how to live with low calories, low fat but nobody talks about over coming hunger.

We have heard how it may be gut bacteria, running low on energy through energy locked in due to insulin, how it may be brain chemistry, temptation, one of the phases of insulin, yet I am able to go a few days without hunger, and in extreme cut periods, it is less of an issue. If I am busy, although being hungry, I am more able to not eat.

I have explored sugar issues, grains, dairy, eggs, starches, Omega 6 oils, fasting, eat everything concepts, and have still found no solutions.

Perhaps it is time to explore the mind as a solution. Sitting, in a hungry state, has been suggested by a eastern grue, and watching the hunger. That is pure torture. Mono meals, one food only has been suggested, but "I can't get no satisfaction." Perhaps it is too weak of satiety signal. Is there any food that boost the signal?

Dr, Jay suggests it is dopamine issue. There is no available treatment.

I have tried supplements, and some had perhaps minor effects. Phen-fen worked for a three day on, 4 day off cycle, but it is not available and dangerous. But even that danger was likely better than the alternative of obesity or starving.

Low carb helps, but even without carb creep, the hunger is there, getting worse each day, each month, each year. And the medical community has no solutions, other than mutilation, and then only about 25 percent keep the weight off.

There is something I am missing, but then I have been obese all my life, losing or gaining, never stable.

hunger

Is hunger the sensation of the switch in the liver from glycogenesis to glyconeogenesis, or perhaps gut fauna signaling. It has nothing to do with the stomach as removal leaves the sensation in some people.

If that is the case, n=1 potato should relieve hunger quickly, and last for a while, as it does. Is the effect lasting? 

Social Delusion

Being hunger free for life is a social delusion, not real. It is my expatiation that I will be hungry the remainder of my life, if I am to maintain this weight loss.

To day Yoni of Weight Matters discusses a paper that indicated that change in lifestyle, work, is the cause of obesity (more non physical work). Work, exercise raises BG through glucogenesis (from glycogen) and gluconegenesis (from protein). Keytones from fat are also produced, for muscle food. Hunger is sometime related to BG, and light exercise reduces hunger for a while, until glycogen storage is reduced. Duh. Backwards.

Now it is a mater of adjusting the human organism to its new environment. We can obtain some relief by returning to old food sources, modern paleo, archevore, primal diet. Once I consider myself as a symbonient mind-body, it is easier to see that hunger is just a body feeling and can be ignored or manipulated. freedom from hunger is just a social delusion pushed by others.

There is a great flow chart at http://www.carbohydratescankill.com/2093/alcohol-hypertriglyceridemia-beyond , but without division at the multiparty split points. Add Lustig, and fructose goes mainly to fats as storage, and fruit and alcohol look similar.

hunger and satiety

from http://zeroinginonhealth.com/Hunger.html Hunger and Satiety

Many people observe that when they eat foods high in protein and fat, they have a hard time binging. However, with carbohydrates, we don’t seem to have that protection. Many of us can imagine eating an entire container of movie popcorn with the butter, or finishing off a dozen donuts or even an entire pizza. Researchers have studied the metabolic background of hunger and they made two important universal observations: First, it’s possible to still be hungry on 10,000 calories of carbohydrates; and, Second, that obese people tend to prefer carbohydrates more than lean individuals. University of Vermont researcher Ethan Sims conducted overfeeding experiments on convicts. He found that a person can eat as much as 10,000 calories per day of mostly carbohydrates and still feel hunger late in the day. Subjects fed 800 superfluous calories of fat developed marked anorexia. Sims and his colleagues neglected to publish the fact that it seemed impossible to fatten their subjects on high-fat, high-protein diets. Edward Horton, who was a colleague of Sims at the time, related that volunteers would sit staring at “plates of pork chops a mile high and they would refuse to eat enough of this meat to constitute the excess thousand calories a day that the Vermont investigators were asking of them. Elliot Danforth worked along with Sims and stated, “The bottom line is that you cannot gain weight on the Atkins diet. It’s just too hard. I challenge anyone to do an overfeeding study with just meat. You can’t do it. I think it’s a physical impossibility.” On a more familiar level, one can imagine eating a large 20-ounce bag of movie popcorn (over 1100 calories) but not 1100 calories of cheese or even a cup and a half of melted Brie. Both the anticipation of carbohydrates and the actual eating of them cause our bodies to secrete the hormone insulin which serves to deposit both fatty acids and glucose in fat tissue and it keeps those calories trapped in fat tissue once they get there. As long as we respond to carbohydrates by secreting more insulin, we continue to move nutrients from our bloodstream in expectation of the arrival of more, so we remain hungry or at least not fulfilled. It’s not that fat fills us up; rather, carbohydrates prevent satiety. For the obese, these are profound implications. Whenever insulin is in circulation, fatty acids are not. Obesity results when fatty acids remain stored longer than they should. Fatty acids move constantly throughout our bodies in the bloodstream. They never sit idle as if they were in a trash can. This has been known since 1948. They move in the bloodstream constantly changing directions responding to the requests for fuel from the organs, muscles and tissues. However, when lipids get trapped in fat tissue, this causes fatty acids to stay in fat tissue for longer than normal and this results in a slowdown. This metabolic slowdown results in weight gain over time. We notice the effect of hyperinsulinemia at restaurants after eating a large steak. We are particularly stuffed yet when the wait staff comes and offers a delectable treat, we seem to find room even though we know we’re stuffed. If the wait staff were to offer us more steak, we would likely refuse. The anticipation of the sweet causes insulin to be released, which lowers our blood sugar, and creates the very real need to balance the blood sugar with more sugar. This is the vicious cycle of hyperinsulinemia and this is present even in lean people. The second observation is that obese people crave carbohydrates in two ways. In the first case, we have insulin secretion during a meal. In the second, we have the usual insulin responses in anticipation of and as a result of the meal. In both cases, insulin induces hunger or prevents satiety. In the case of hyperinsulinemia, this happens even between meals and/or during the night when the cells should be living off a fuel mixture predominantly of fatty acids. Instead, the high insulin traps the fatty acids in fat tissue and primes the cells to take up glucose in place of oxygen. As far as the body is concerned, this elevated insulin indicates that we’ve just eaten. However, in this case, we haven’t. Our homeostatic system is expecting to deal with glucose but there is none coming in the diet. Despite this there is still too much insulin in the circulation. The liver will not give up its glycogen due to high insulin in circulation and our blood sugar drops causing us to crave glucose. Even if we eat fat and protein, the hyperinsulinemia will serve to store those nutrients rather than allow them to be used for fuel. The implication is critical to how we perceive the dietary treatment of obesity and one in which even the current low-carbohydrate diet plans do not address. The craving for carbohydrates is a physiological one heralded by high insulin. Any diet that fails to control both cravings and hunger is sure to fail. The craving for carbohydrates is more closely akin to an addiction as British clinician Robert Kemp described it in 1963. Carbohydrates cause Hyperinsulinemia through the diet creating an addiction similar to nicotine or cocaine or any other addictive substance. There is nothing inherently natural about these addictions. Te hunger that goes along with semi-starvation is an unavoidable condition. The craving for carbohydrates is not. Sugar provides an exaggerated response to the region of the brain known as the reward center, the nucleus accumbens. Intense cravings for sugar may be explained by the intensity of the dopamine secretion in the brain when sugar is consumed. When the nucleas accubens is excessively activated by sweets or powerful drugs, it leads to abuse or addiction. When the system is underactive, it results in signs of depression. The fact that this is an addiction indicates that it can be overcome with sufficient time, effort, and motivation and this is not the case with hunger itself (except in the case of the anorexic). Avoiding carbohydrates will lower insulin over time and so ameliorate the hyperinsulinemia that causes the carbohydrate craving itself. Dr. James Sidbury found that after a year to 18 months the appetite is normalized and the craving for sweets is lost. Carbohydrate restriction is very difficult but with sufficient time and motivation, a person can indeed overcome their addiction to the sweet and thus improve their health. To overcome this addiction to carbohydrates, will require support. There isonline support available at the Forum where you can come and learn what it's like for someone with a zero-carb lifestyle.