Monday, November 21, 2011

Comments to others

So now have insulin resistance causes obesity, insulin resistance is caused by overeating. Then once we become insulin resistance, aka overfed, we have a physical desire to maintain that overfed state as long as food is pleasureful and available. OK. More ELMM advise comming?

There are so many things that do not apply to a “damaged metabolism” here.

A day of "overeating" will start insulin resistance. Once insulin resistance is started, our appetite expands to keep the insulin resistance there. Probably the thrift gene type effect.  Now we are on a cycle of weight gain, insulin resistance, increased appetite, overeating.

Insulin resistance is the body's way of distributing energy, first overflowing the liver's storage capacity, then muscles, and into fat. When fat cannot handle it, you got a major problem. 

Consuming an ample supply of palatable food is sufficient to start insulin resistance, and unless we make a conscious effort to cut the next day, we will be into increased appetite. Then there all those other things we eat over, social pressure, anxiety, boredom. Once we start, our appetite increases. 

Insulin and insulin resistance is just the body's way of dealing with the process. It is our nature to eat /overeat when food is available. With full time processed food, and no great need for energy expenditure,  what we can expect:  our nature, to do what it does. 

On your graph, to simply see the effect of overeating, shift your zero axis downward or tilt to the right to see the accumulative effect of overeating, which must occur for weight gain.    

To remain non-obese, we must avoid eating processed food, and overeating which is against our vary nature. We must overcome our food seeking nature.

And to GT, one off the wall idea. I suspect that the reason that many of us have greater weight accumulation in the lower body is due to laminar blood flow directing slightly greater concentration of insulin and nutrients to the lower portion of the body and the first pass effect. Or perhaps it is our lower body gets a greater volume of nutrition per unit of mass than the remainder.

I accept that these people do not read my blog, but I have said my piece. 

Also in order to beat the glucose challenge, we must train our bodies to up-regulate.  Is the glucose bolus a fit way to test for dietetics in a low-carber?  A1c  Ok, but after a potato with supper, after a week of VLC, my AM glucose is in diabetic range.

And what do I know? 

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